in-house developed code Search Results


pls toolbox 8.8  (Eigenvector Research Inc)
90
Eigenvector Research Inc pls toolbox 8.8
Pls Toolbox 8.8, supplied by Eigenvector Research Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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pls toolbox 8.8 - by Bioz Stars, 2026-04
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matlab code r2019a  (MathWorks Inc)
90
MathWorks Inc matlab code r2019a
Matlab Code R2019a, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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matlab code r2019a - by Bioz Stars, 2026-04
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musculoskeletal simulation code cobi-dyn  (CFD Research Corporation)
90
CFD Research Corporation musculoskeletal simulation code cobi-dyn
Musculoskeletal Simulation Code Cobi Dyn, supplied by CFD Research Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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musculoskeletal simulation code cobi-dyn - by Bioz Stars, 2026-04
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matlab code 2017b  (MathWorks Inc)
90
MathWorks Inc matlab code 2017b
Matlab Code 2017b, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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matlab code 2017b - by Bioz Stars, 2026-04
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inhouse code  (MathWorks Inc)
90
MathWorks Inc inhouse code
A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in <t>MATLAB.</t> Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.
Inhouse Code, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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inhouse code - by Bioz Stars, 2026-04
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a polyclonal antibody (code no. 95234) raised in rabbits against human gip(1-30)nh2  (Phoenix Pharmaceuticals)
90
Phoenix Pharmaceuticals a polyclonal antibody (code no. 95234) raised in rabbits against human gip(1-30)nh2
A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in <t>MATLAB.</t> Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.
A Polyclonal Antibody (Code No. 95234) Raised In Rabbits Against Human Gip(1 30)Nh2, supplied by Phoenix Pharmaceuticals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/a polyclonal antibody (code no. 95234) raised in rabbits against human gip(1-30)nh2/product/Phoenix Pharmaceuticals
Average 90 stars, based on 1 article reviews
a polyclonal antibody (code no. 95234) raised in rabbits against human gip(1-30)nh2 - by Bioz Stars, 2026-04
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matlab r2019b  (MathWorks Inc)
90
MathWorks Inc matlab r2019b
A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in <t>MATLAB.</t> Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.
Matlab R2019b, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/matlab r2019b/product/MathWorks Inc
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matlab r2019b - by Bioz Stars, 2026-04
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c++ module  (MathWorks Inc)
90
MathWorks Inc c++ module
A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in <t>MATLAB.</t> Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.
C++ Module, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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c++ module - by Bioz Stars, 2026-04
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matlab code r2020b  (MathWorks Inc)
90
MathWorks Inc matlab code r2020b
A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in <t>MATLAB.</t> Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.
Matlab Code R2020b, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/matlab code r2020b/product/MathWorks Inc
Average 90 stars, based on 1 article reviews
matlab code r2020b - by Bioz Stars, 2026-04
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matlab script v7.1  (MathWorks Inc)
90
MathWorks Inc matlab script v7.1
A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in <t>MATLAB.</t> Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.
Matlab Script V7.1, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/matlab script v7.1/product/MathWorks Inc
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matlab script v7.1 - by Bioz Stars, 2026-04
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matlab r2012b  (MathWorks Inc)
90
MathWorks Inc matlab r2012b
A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in <t>MATLAB.</t> Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.
Matlab R2012b, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/matlab r2012b/product/MathWorks Inc
Average 90 stars, based on 1 article reviews
matlab r2012b - by Bioz Stars, 2026-04
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in-house code  (MathWorks Inc)
90
MathWorks Inc in-house code
A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in <t>MATLAB.</t> Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.
In House Code, supplied by MathWorks Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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in-house code - by Bioz Stars, 2026-04
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Image Search Results


A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in MATLAB. Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.

Journal: bioRxiv

Article Title: Mechanical stress in pancreatic cancer: Signaling pathway adaptation activates cytoskeletal remodeling and enhances cell migration

doi: 10.1101/2021.06.11.448065

Figure Lengend Snippet: A, MIA PaCa-2 and PANC-1 cells were pre-treated with 15 μM p38 MAPK inhibitor (SB202190), JNK inhibitor (SP600125) or equal volume of DMSO and subjected to a scratch wound healing assay under 4.0 mmHg of compression. Pictures from at least 4 different fields were taken from three biological replicates. Scale bar: 0.2 mm. White dashed line shows the difference in wound closure between 0 and 16 hours. B-C, Graph showing the average ±SE percentage wound closure of compressed MIA PaCa-2 (B) and PANC-1 (C) treated with DMSO or inhibitors from 3 biological replicates (n≥12). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). D-E , Graph showing the average Ki67 area fraction in compressed MIA PaCa-2 (D) and PANC-1 (E) cells treated with DMSO, p38 MAPK or JNK inhibitor from at least 10 different fields/ condition from two biological replicates as quantified automatically using an in-house code in MATLAB. Asterisk (*) indicates a statistically significant difference in student’s t test (p<0.05). F , Representative Western Blotting showing phosphorylated HSP27 (Ser 82) and c-Jun (S73) in control and compressed MIA PaCa-2 cells treated with 15 μM of each inhibitor or equal volume of DMSO. Antibody against β-actin was used as a loading control. Quantification of each antibody compared to loading control was quantified by ImageJ and it is indicated by numbers in grey font.

Article Snippet: For image analysis, the calculation of Ki67 area fraction was performed automatically using a previously developed inhouse code in MATLAB (MathWorks, Inc., Natick,MA) , , .

Techniques: Wound Healing Assay, Western Blot, Control

A-B , Rac1 (A) and cdc42 (B) mRNA expression was quantified by qPCR in both MIA PaCa-2 and PANC-1. Each bar indicates the mean fold change ±SE of two biological replicates (n=6). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). C , MIA PaCa-2 and PANC-1 pancreatic cancer cells were treated with siRNA against Rac1 (siRac1) or cdc42 (sicdc42) and then subjected to a scratch wound healing assay for 16 hours under 0.0 or 4.0 mmHg of compression in 2 % FBS containing DMEM. Control cells were treated with stealth siRNA (siCTRL). Scale bar: 0.1 mm. D-E , Graphs showing the percentage ±SE wound closure of MIA PaCa-2 (D) and PANC-1 (E) as quantified using ImageJ software. Statistically significant difference in wound closure of compressed siRac1- or sicdc42-treated cells compared to siCTRL-treated cells is indicated with an asterisk (*) (2 biological replicates; n≥6; p<0.05 in student’s t test). F , Graph showing the average ±SE Ki67 area fraction in MIA PaCa-2 and PANC-1 control and compressed cells treated with siCTRL, siRac1 or sicdc42 from at least 5 different fields/ condition from two biological replicates as quantified automatically using an in-house code in MATLAB. No statistically significant changes were observed.

Journal: bioRxiv

Article Title: Mechanical stress in pancreatic cancer: Signaling pathway adaptation activates cytoskeletal remodeling and enhances cell migration

doi: 10.1101/2021.06.11.448065

Figure Lengend Snippet: A-B , Rac1 (A) and cdc42 (B) mRNA expression was quantified by qPCR in both MIA PaCa-2 and PANC-1. Each bar indicates the mean fold change ±SE of two biological replicates (n=6). Asterisk (*) indicates a statistically significant difference (p<0.05 in student’s t test). C , MIA PaCa-2 and PANC-1 pancreatic cancer cells were treated with siRNA against Rac1 (siRac1) or cdc42 (sicdc42) and then subjected to a scratch wound healing assay for 16 hours under 0.0 or 4.0 mmHg of compression in 2 % FBS containing DMEM. Control cells were treated with stealth siRNA (siCTRL). Scale bar: 0.1 mm. D-E , Graphs showing the percentage ±SE wound closure of MIA PaCa-2 (D) and PANC-1 (E) as quantified using ImageJ software. Statistically significant difference in wound closure of compressed siRac1- or sicdc42-treated cells compared to siCTRL-treated cells is indicated with an asterisk (*) (2 biological replicates; n≥6; p<0.05 in student’s t test). F , Graph showing the average ±SE Ki67 area fraction in MIA PaCa-2 and PANC-1 control and compressed cells treated with siCTRL, siRac1 or sicdc42 from at least 5 different fields/ condition from two biological replicates as quantified automatically using an in-house code in MATLAB. No statistically significant changes were observed.

Article Snippet: For image analysis, the calculation of Ki67 area fraction was performed automatically using a previously developed inhouse code in MATLAB (MathWorks, Inc., Natick,MA) , , .

Techniques: Expressing, Wound Healing Assay, Control, Software